Regular ArticleRegulation by Interleukin-1β of Gene Expression of Bradykinin B1 Receptor in MH-S Murine Alveolar Macrophage Cell Line
References (39)
- et al.
Trends Neurosci.
(1993) - et al.
Eur. J. Pharmacol.
(1981) - et al.
Immunopharmacology
(1989) - et al.
Eur. J. Pharmacol.
(1994) - et al.
FEBS Lett.
(1989) Trends Pharmacol. Sci.
(1988)- et al.
J. Biol. Chem.
(1994) - et al.
Immunopharmacology
(1996) - et al.
Biochem. Biophys. Res. Commun.
(1996) - et al.
Biochem. Biophys. Acta
(1995)
Biochem. Biophys. Res. Commun.
Eur. J. Pharmacol.
Biochem. Pharmacol.
Life Sci.
J. Biol. Chem.
Annu. Rev. Immunol.
Agents Actions
Can. J. Physiol. Pharmacol.
Br. J. Pharmacol.
Cited by (21)
Activation of peritoneal macrophages during the evolution of type 1 diabetes (insulitis) in streptozotocin-treated mice
2010, PeptidesCitation Excerpt :It appears that in our control mice the B1 receptors are present but in small quantities. Reports from other groups have also indicated that rodent monocytes could express the B1 receptor [4,21,22]. Our results also showed that LPS released much larger amounts (up to 3 times) of NO from macrophages obtained from the three groups of diabetic animals than from control ones.
Kinin B1 and B2 receptor expression in osteoblasts and fibroblasts is enhanced by interleukin-1 and tumour necrosis factor-α. Effects dependent on activation of NF-κB and MAP kinases
2008, BoneCitation Excerpt :It is well documented that IL-1β and TNF-α can up-regulate several biological responses to B1 receptor agonists in a variety of cell types. Direct evidence for that these responses, to IL-1β and TNF-α, are associated with increased B1 receptor expression, assessed by either binding assays or by mRNA analysis, have previously been demonstrated in the human lung fibroblastic cell lines IMR-90, HEL299 and WI-38 [14,36–38,41–43], the human lung epithelial cell line A-549 [37,44], in the MH-S murine alveolar macrophage cell line [45] and in primary rabbit aortic [23] and rat vascular [34] smooth muscle cell. Increased mRNA expression and binding of B2 receptors by IL-1β and TNF-α have previously only been shown in HEL299 and A-549 cell lines [14,44].
p38 stress-activated protein kinase inhibitor reverses bradykinin B<inf>1</inf> receptor-mediated component of inflammatory hyperalgesia
2001, European Journal of PharmacologyKinin B<inf>1</inf> receptors and the cardiovascular system: Regulation of expression and function
2000, Cardiovascular Research
- 1
To whom correspondence should be addressed at First Department of Internal Medicine, Gunma University School of Medicine, 3-39-15 Showa Maebashi, Gunma, 371-8511, Japan. Fax: 027-220-8136. E-mail: [email protected].