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"The unique α4(+)/(-)α4 agonist binding site in (α4)3(β)2 subtype nicotinic acetylcholine receptors permits differential agonist desensitization pharmacology vs. the (α4)2(β2)3 subtype." Journal of Pharmacology and Experimental Therapeutics
(2013):
jpet.113.208389.
Web. 20 April. 2024.