… berghei parasite and of GFP from another never leads to signal overlap in dually infected
mouse erythrocytes (JM Matz, personal communication). Furthermore, the vacuole does not …

During intraerythrocytic growth, the human malaria parasite Plasmodium falciparum degrades
up to 80% of the host cell's hemoglobin inside an acidified organelle called the digestive …

The accumulation of heat shock proteins (HSPs) after the exposure of cells or organisms to
elevated temperatures is well established. It is also known that a variety of other …

Protein export is considered an essential feature of malaria parasite blood stage development.
Here, we examined five components of the candidate Plasmodium translocon of exported …

Plasmodium berghei was identified as a parasite of thicket rats (Grammomys dolichurus) and
Anopheles dureni mosquitoes in African highland forests. Successful adaptation to a range …

To ensure their survival in the human bloodstream, malaria parasites degrade up to 80% of
the host erythrocyte hemoglobin in an acidified digestive vacuole. Here, we combine …

The erythrocyte is an extraordinary host cell for intracellular pathogens and requires extensive
remodelling to become permissive for infection. Malaria parasites modify their host red …

During blood-stage development, malaria parasites are challenged with the detoxification of
enormous amounts of heme released during the proteolytic catabolism of erythrocytic …

Recent reports indicate that Heat Shock Proteins (HSPs) are induced in mammalian tissues
as part of a homeostatic response to environmental stressors. Administration of …

Malaria pathology is linked to remodeling of red blood cells by eukaryotic Plasmodium parasites.
Central to host cell refurbishment is the trafficking of parasite-encoded virulence factors …