Acetoacetate increases expression of intercellular adhesion molecule-1 (ICAM-1) in human brain microvascular endothelial cells
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Acknowledgements
The study was supported by the VA Medical Research Service. The authors acknowledge the expert secretarial assistance of Ms C. Masters.
References (25)
- et al.
NF-Kappa B is activated and ICAM-1 gene expression is upregulated during reoxygenation of human brain endothelial cells
Neurosci. Lett.
(1998) - et al.
Acetoacetate and beta-hydroxybutyrate differentially regulate endothelin-1 and vascular endothelial growth factor in mouse brain microvascular endothelial cells
J. Diabetes Complications
(1999) - et al.
Expression of intercellular adhesion molecule-1 induced by high glucose concentrations in human aortic endothelial cells
Life Sci.
(2001) Mitochondrial redox state in the critically ill
Br. J. Anaesth.
(1999)- et al.
Plasma concentrations of VCAM-1 and ICAM-1 are elevated in patients with Type 1 diabetes mellitus with microalbuminuria and overt nephropathy
Diabet. Med.
(2000) - et al.
Correlates of brain edema in uncontrolled IDDM
Diabetes
(1992) - et al.
Induction of IL-1 and interferon-Y: tissue distribution, biochemistry, function of a natural adherence molecule (ICAM-1)
J. Immunol.
(1986) - et al.
Abnormal markers of endothelial cell activation and oxidative stress in children, adolescents and young adults with Type 1 diabetes with no clinical vascular disease
Diabetes Metab. Res. Rev.
(1999) Use of anti-ICAM-1 therapy in ischemic stroke: results of the enlimomab acute stroke trial
Neurology
(2001)- et al.
Hypertonic mannitol loading of NF-κB transcription factor decoys in human brain microvascular endothelial cells blocks upregulation of ICAM-1
Stroke
(2000)
Cranial computed tomography in children and adolescents with diabetic ketoacidosis
Am. J. Neuroradiol.
(1988)
Hyperketonemia can increase lipid peroxidation and lower glutathione levels in human erythrocytes in vitro and in Type 1 diabetic patients
Diabetes
(1999)
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