Memantine, but not dizocilpine, ameliorates cognitive deficits in adult rats withdrawn from chronic ingestion of alcohol
Section snippets
Acknowledgements
This work was supported by grants from Fundação para a Ciência e a Tecnologia (FCT) to Research Unit 121/94 and to N.V.L. (BFRH/BPD/1583/2000).
References (22)
- et al.
Neuroprotective concentrations of the N-methyl-D-aspartate open-channel blocker memantine are effective without cytoplasmic vacuolation following post-ischemic administration and do not block maze learning or long-term potentiation
Neuroscience
(1998) - et al.
Increased NMDA-induced excitability during ethanol withdrawal: a behavioural and histological study
Brain Res.
(1995) - et al.
Effects of baclofen and nitrendipine on ethanol withdrawal responses in the rat
Neuropharmacology
(1991) - et al.
Ethanol withdrawal seizures and the NMDA receptor complex
Eur. J. Pharmacol.
(1990) Maze procedures: the radial-arm and water maze compared
Cogn. Brain Res.
(1996)- et al.
MK-801-induced neuronal damage in rats
Brain Res.
(1997) The competitive NMDA receptor antagonist, CGP39551, inhibits ethanol withdrawal seizures
Eur. J. Pharmacol.
(1991)- et al.
Synaptic reorganization in the hippocampal formation of alcohol-fed rats may compensate for functional deficits related to neuronal loss
Alcohol
(2000) - et al.
Behavioral and neuroanatomical consequences of chronic ethanol intake and withdrawal
Physiol. Behav.
(1999) - et al.
A single high dose of dizocilpine produces long-lasting impairment of the water maze performance in adult rats
Neurosci. Lett.
(2000)
Developments of a water-maze procedure for studying spatial learning in the rat
J. Neurosci. Methods
(1984)
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