Chapter 4 Cell death in models of spinal cord injury
References (91)
- et al.
Glutamate-induced neuronal death: a succession of necrosis or apoptosis depending on mitochondrial function
Neuron
(1995) - et al.
Impaired mitochondrial function, oxidative stress and altered antioxidant enzyme activities following traumatic spinal cord injury
Brain Res.
(1997) - et al.
Does oligodendrocyte survival depend on axons?
Curr. Biol.
(1993) - et al.
Endogenous repair after spinal cord contusion injuries in the rat
Exp. Neural.
(1997) - et al.
Cell death and plasticity after experimental spinal cord injury
- et al.
A behavioral and morphological analysis of spinal cord injuries produced by a feedback controlled impact device
Exp. Neurol.
(1987) - et al.
Oligodendroglial apoptosis occurs along degenerating axons and is associated with Fas and p75 expression following spinal cord injury in the rat
Neuroscience
(2001) - et al.
Neurotrophin receptors: mediators of life and death
Brain Res. Rev.
(1998) Glutamate receptors and the induction of excitotoxic neuronal death
Ischemia-induced neuronal apoptosis
Curr. Opin. Neurobiol.
(1996)
Brain-derived neurotrophic factor in astrocytes, oligodendrocytes, and microglia/macrophages after spinal cord injury
Neurobiol. Dis.
(2000)
The relationships among the severity of spinal cord injury, residual neurological function, axon counts, and counts of retrogradely labeled neurons after experimental spinal cord injury
Exp. Neural.
(1995)
Relationship of altered glutamate receptor subunit mRNA expression to acute cell loss after spinal cord contusion
Exp. Neurol.
(2001)
Tumor necrosis factor-alpha has morphological effects within the dorsal columns of the spinal cord in contrast to its effect in the peripheral nervous system
J. Neuroimmunol.
(2000)
Tumor necrosis factor induces cFOS and strongly potentiates glutamate-mediated cell death in rat spinal cord
Neurobiol. Dis.
(2001)
Degeneration and sprouting of identified descending supraspinal axons after contusive spinal cord injury in the rat
Exp. Neurol.
(2001)
Apoptosis by death factor
Cell
(1997)
Spinal cord contusion in the rat: morphometric analyses of alterations in the spinal cord
Exp. Neurol.
(1985)
Extracellular calcium activity in the injured spinal cord
Exp. Neurol.
(1983)
Increase of interleukin-1 beta mRNA and protein in the spinal cord following experimental traumatic injury in the rat
Brain Res.
(1997)
Delayed glial cell death following Wallerian degeneration in white matter tracts after spinal cord dorsal column cordotomy in adult rats
Exp. Neurol.
(2001)
Amelioration of functional deficits from spinal cord trauma with systemically administered NBQX, an antagonist of non-Nmethyl-d-aspartate receptors
Exp. Neurol.
(1996)
Delayed antagonism of AMPA/kainate receptors reduces long-term functional deficits resulting from spinal cord trauma
Exp. Neurol.
(1997)
Methylprednisolone inhibition of TNF-α expression and NFκB activation after spinal cord injury in rats
Mol. Brain Res.
(1998)
Apoptotic cells associated with Wallerian degeneration after experimental spinal cord injury: a possible mechanism of oligodendroglial death
J. Neurotrauma
(1999)
Caspase-3 inhibition is neuroprotective after spinal cord injury
J. Neurochem.
(2000)
Pathology of experimental spinal cord trauma. I. The necrotic lesion as a function of vascular injury
Lab. Invest.
(1978)
Pathology of experimental spinal cord trauma. II. Ultrastructure of axons and myelin
Lab. Invest.
(1978)
Cell death in the oligodendrocyte lineage
J. Neurobiol.
(1992)
Histological and locomotor studies of graded spinal cord contusion using the NYU weight-drop device versus transection
Exp. Neurol.
(1996)
Cell death, repair, and recovery of function after spinal cord contusion injuries in rats
Expression of p75 NTFR in neurons and oligodendrocytes after spinal cord injury in rats: neurotrophin-induced apoptosis in vivo?
Soc. Neurosci. Abstr.
(1999)
A review of current evidence for apoptosis after spinal cord injury
J. Neurotrauma
(2000)
TNF, glutamate and neurodegeneration in the rat spinal cord
Soc. Neurosci. Abstr
(2000)
Spinal cord injury produced by consistent mechanical displacement of the cord in rats: behavioral and histologic analysis
J. Neurotrauma
(1992)
Systemically administered interleukin-10 reduces tumor necrosis factor-alpha production and significantly improves functional recovery following traumatic spinal cord injury in rats
J. Neurotrauma
(1999)
Efficacy of methylprednisolone in acute spinal-cord injury
J. Am. Med. Assoc.
(1984)
A randomized controlled trial of methylprednisolone or naloxone in the treatment of acute spinal-cord injury
New Engl. J. Med.
(1990)
Administration of methylprednisolone for 24 or 48 hours or tirilizad mesylate for 48 hours in the treatment of acute spinal cord injury — results of the Third National Acute Spinal Cord Injury Randomized Controlled Trial
J. Am. Med. Assoc.
(1997)
Observations of the pathology of human spinal cord injury. A review and classification of 22 new cases with details from a case of cord compression with extensive focal demyelination
Adv. Neurol.
(1993)
An endotoxin-induced serum factor that causes necrosis of tumors
Neurotrophins: the biological paradox of survival factors eliciting apoptosis
Cell Death Differ.
(1998)
The effects of methylprednisolone and the ganglioside GMI on acute spinal cord injury in rats
J. Neurosurg.
(1994)
Morphological evidence suggesting apoptotic nuclei in spinal cord injury
Soc. Neurosci. Abstr.
(1995)
Apoptosis and delayed degeneration after spinal injury in rats and monkeys
Nat. Med.
(1997)
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