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2010, Mayo Clinic ProceedingsEstrogen-induced relaxation of the rat tail artery is attenuated in rats with pulmonary hypertension
2010, Pharmacological ReportsCitation Excerpt :The female hormone estrogen has multiple cardiovascular effects. Apart from genomic and chronic effects, its direct vascular action has been studied for a long time, but there are still controversies about which mechanisms are involved in this action [8, 16]. Recent literature has focused on endothelium-independent mechanisms of estrogen-induced vascular dilation, such as the blocking of calcium channels or activation of Ca2+-activated K+ channels [5, 13, 18].
Sex differences and the effects of sex hormones on hemostasis and vascular reactivity
2001, Heart and Lung: Journal of Acute and Critical CareCitation Excerpt :One is related to VSMC sensitivity to norepinephrine that may be modulated by testosterone, and the other is related to adrenergic nerve stimulation and neurotransmitter release and uptake that may be influenced by ovarian hormones. A study conducted by Garcia-Villalon et al,183 whose findings were similar to those of Li et al,182 also used the isolated rat tail artery to show that adrenergic nerve stimulation caused greater vasoconstriction in male arteries compared with female arteries. However, Garcia-Villalon and colleagues measured the amount of norepinephrine released during nerve stimulation and showed it did not differ between males and females.
Effects of chronic ethanol consumption on aortic constriction in male and female rats
1999, European Journal of PharmacologySympathetic nerve activity, stress, and cardiovascular risk
2016, Handbook of PsychocardiologyReduced vasorelaxation to estradiol and G-1 in aged female and adult male rats is associated with GPR30 downregulation
2013, American Journal of Physiology - Endocrinology and Metabolism