Special sectionCocaine use in pregnancy: Perinatal morbidity and mortality☆
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Cited by (247)
Paternal and/or maternal preconception-induced neurobehavioral teratogenicity in animal and human models
2021, Brain Research BulletinCitation Excerpt :The recovery period after biomass smoke exposure seems to have an effect on the neurobehavioral outcomes observed in the offspring. Cocaine is a teratogen known to affect the dopaminergic system and its use prenatally has been shown to cause growth and behavioral abnormalities (Chasnoff et al., 1987, 1985; Dow-Edwards, 1989) As reported in a subsequent review (Nelson et al., 1996), paternal exposure to cocaine led to hyperactivity and impaired learning and memory (spontaneous alternation test) in the offspring of exposed rats (Abel, 1989).
Passive Addiction and Teratogenic Effects
2018, Volpe's Neurology of the NewbornEffects of state contraceptive insurance mandates
2017, Economics and Human BiologyDelivery room management of meconium-stained infant
2012, Clinics in PerinatologyCitation Excerpt :Fig. 1 describes the various conditions and the possible mechanisms for MSAF. Other factors associated with meconium passage are drug abuse,19 intrauterine infection,20 use of vaginal misoprostol for labor induction,21 and gestational cholestasis.22 In the latter instance, MSAF was attributed to an increased fetal colonic stimulation by higher bile acid accumulation in the maternal serum.
Maternal Drug Abuse: Effects on the Fetus and Neonate
2011, Fetal and Neonatal Physiology E-Book, Fourth EditionCocaine, hormones, and behavior: Clinical and preclinical studies
2009, Hormones, Brain and Behavior Online
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Supported by a grant from the National Institute on Drug Abuse, NIH (DA04103-01).