Relationship between the development of behavioral tolerance and the biodisposition of phencyclidine in mice☆
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Cited by (10)
Disruption of Performance in the Five-Choice Serial Reaction Time Task Induced By Administration of N-Methyl-D-Aspartate Receptor Antagonists: Relevance to Cognitive Dysfunction in Schizophrenia
2010, Biological PsychiatryCitation Excerpt :Similarly, the exacerbation of schizophrenia symptoms after NMDA antagonist administration includes worsening of cognitive symptoms (50). NMDA antagonist administration also disrupts cognitive performance in experimental animals, including rats, mice, and monkeys (25,60–108,109–137). The best exposure regimen for inducing cognitive deficits with NMDA antagonists in experimental animal models has been a subject of some debate.
Evidence for impairment of behavioural inhibition in performance of operant tasks in tPA-/- mice
2001, Behavioural Brain ResearchEffects of repeated phencyclidine treatment on serotonin transporter in rat brain
2000, Neuroscience LettersPharmacological potency and biodisposition of phencyclidine via inhalation exposure in mice
1996, Drug and Alcohol DependenceDiscriminative stimulus properties and brain distribution of phencyclidine in rats following administration by injection and smoke inhalation
1985, Pharmacology, Biochemistry and Behavior
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Supported by NIDA Grants DA-01442 and DA-02396.
- 2
Pre-doctoral Fellow supported by NIDA Grant DA-07027.
Copyright © 1984 Published by Elsevier Inc.