Inhibition of platelet thromboxane A2 synthase activity by sodium 5-(3′-pyridinylmethyl)benzofuran-2-carboxylate
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Central bombesin activates adrenal adrenaline- and noradrenaline-containing cells via brain thromboxane A<inf>2</inf> in rats
2009, Autonomic Neuroscience: Basic and ClinicalCentrally administered neuromedin U elevates plasma adrenaline by brain prostanoid TP receptor-mediated mechanisms in rats
2008, European Journal of PharmacologyAdrenal adrenaline- and noradrenaline-containing cells and celiac sympathetic ganglia are differentially controlled by centrally administered corticotropin-releasing factor and arginine-vasopressin in rats
2007, European Journal of PharmacologyCitation Excerpt :The present results are well consistent with those of our previous report in which central pretreatment with indomethacin abolished the centrally administered CRF- and AVP-induced elevation of plasma catecholamines in rats (Yokotani et al., 2001; Okada et al., 2003a). Finally, we examined the effects of furegrelate, a selective inhibitor of thromboxane A2 synthase (Gorman et al., 1983), on the centrally administered CRF- and AVP-induced cFos expression. Central pretreatment with furegrelate abolished the CRF-induced cFos expression in the adrenal A-cells (but not noradrenergic neurons in the celiac ganglia), and also abolished the AVP-induced all cFos expression in the adrenal A-cells and NA-cells.
Brain prostanoid TP receptor-mediated adrenal noradrenaline secretion and EP<inf>3</inf> receptor-mediated sympathetic noradrenaline release in rats
2005, European Journal of Pharmacology
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