Priming of experimental anxiety by repeated subthreshold GABA blockade in the rat amygdala
References (41)
- et al.
Temporal lobe abnormalities in panic disorder: an MRI study
Biol. Psychiatry
(1990) - et al.
Cardiovascular responses elicited by stimulation of neurons in the central amygdaloid nucleus in awake but not anesthetized rats resemble conditioned emotional responses
Brain Res.
(1987) - et al.
Infusion of the non-NMDA receptor antagonist CNQX into the amygdala blocks the expression of fear-potentiated startle
Behav. Neural Biol.
(1993) Neuroelectrical activity related to panic disorder
Prog. Neuro-Psychopharmacol. Biol. Psychiatry
(1990)- et al.
EEG and CT findings in patients with panic disorder
Biol. Psychiatry
(1990) - et al.
Anti-anxiety action of diazepam after intra-amygdaloid application in the rat
Neuropharmacology
(1979) - et al.
Evidence that the anticonflict effect of midazolam in amygdala is mediated by the specific benzodiazepine receptors
Neurosci. Lett.
(1985) Modification of seizure activity by electrical stimulation: II. Motor seizure
Electroencephalogr. Clin. Neurophysiol.
(1972)- et al.
Blockade of GABAA receptors in the region of the anterior basolateral amygdala of rats elicits increases in heart rate and blood pressure
Brain Research
(1991) - et al.
Anxiolytic effects of chlorodiazepoxide blocked by injection of GABAA and benzodiazepine receptor antagonists in the region of the anterior basolateral amygdala of rats
Biol. Psychiatry
(1995)
Anticonflict effect of the benzodiazepines mediated by a gabaergic mechanism in the amygdala
Eur. J. Pharmac.
PET in generalized anxiety disorder
Biol. Psychiatry
Ideational components of anxiety neurosis
Arch. Gen. Psych.
Epilepsy and Psychiatry
Mental symptoms in temporal lobe epilepsy and temporal lobe gliomas
Acta Psychiatr. Neuro. Scand. (Suppl. 120)
Refractory anxiety: a masked epileptiform disorder?
Psychiatr. J. Univ. Ottawa
Intra-amygdala infusion of theN-methyl-D-aspartate receptor antagonist AP5 blocks acquisition but not expression o f fear-potentiated startle to an auditory conditioned stimulus
Behav. Neurosci.
Extinction of fear-potentiated startle: blockade by infusion of an NMDA antagonist into the amygdala
J. Neurosci.
Localization of discharge in temporal lobe automatism
Arch. Neurol. Psych.
Anxiety precipitated by lactate
N. Engl. J. Med.
Cited by (63)
Exposure of male mice to perfluorooctanoic acid induces anxiety-like behaviors by increasing corticotropin-releasing factor in the basolateral amygdala complex
2022, ChemosphereCitation Excerpt :The powerful GABAergic inhibitory circuit in the BLA is very important in controlling motion and attention behavior (Martijena et al., 2002). The removal of tonic inhibition in the BLA by local infusion of GABA antagonists in vivo results in anxiety-like behaviors (Sanders et al., 1995). An increase in the excitability of BLA neurons by acute CRF receptor activation has been proposed to be a critical molecular component of anxiety-like behavior (Walker and Davis, 2008).
Stress, Panic, and Central Serotonergic Inhibition
2017, Stress: Neuroendocrinology and NeurobiologyThe recovery of acetylcholinesterase activity and the progression of neuropathological and pathophysiological alterations in the rat basolateral amygdala after soman-induced status epilepticus: Relation to anxiety-like behavior
2014, NeuropharmacologyCitation Excerpt :Some of the victims of the Tokyo sarin attacks, for example, presented posttraumatic stress disorder, five or seven years after the events (Hoffman et al., 2007). Hyperexcitability of the amygdala, and the BLA in particular, is a characteristic feature of anxiety-like behavior in animals (Bueno et al., 2005; Davis et al., 1994; Sanders et al., 1995) and anxiety disorders in humans (Koenigs and Grafman, 2009; Rauch et al., 2000; Villarreal and King, 2001). Therefore, the increased anxiety in animals and humans after nerve agent exposure is likely to be due to neuropathological and pathophysiological alterations that have increased the excitability of the BLA network.
The most common native medicinal plants used for psychiatric and neurological disorders in Urmia city, northwest of Iran
2014, Asian Pacific Journal of Tropical DiseaseChronic mild stress-induced changes of risk assessment behaviors in mice are prevented by chronic treatment with fluoxetine but not diazepam
2014, Pharmacology Biochemistry and Behavior
Preliminary results of this study were presented at the 1992 annual meeting of the Society for Neuroscience at New Orleans, LA.