Sympathetic inhibition and attenuation of spontaneous hypertension by PVN lesions in rats
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2020, European Journal of PharmacologyCitation Excerpt :After a 7-day recovery period, glucose or saline in a volume of 0.3 μl was injected into the PVN over 30 s and was left in place for an additional 30 s. The method of the PVN lesion is similar to previous reports (Takeda et al., 1991; Chaleek et al., 2012). Briefly, the PVN were lesioned bilaterally by passing a 2-mA direct current for 10 s. Coordinates of a concentric bipolar stainless steel electrode insulated to within 0.5 mm of the tip were as follows: lateral: ± 0.4 mm from midline; dorsoventral: 8 mm from skull surface; anteroposterior: 1.8 mm from the bregma.
Does the median preoptic nucleus contribute to sympathetic hyperactivity in spontaneously hypertensive rats?
2016, Autonomic Neuroscience: Basic and ClinicalCitation Excerpt :Hence, it is conceivable that MnPO could modulate, through its PVN connections, the pathways that are involved in sympathetic outflow, and consequently the cardiovascular function. Based on these evidences (Allen, 2002; Takeda et al., 1991), it is conceivable that the inhibition of MnPO could decrease the activity of PVN and RVLM neurons which subsequently result in decreases in the RSNA and MAP. In fact, in the present study, we observed that the inhibition of the MnPO caused decrease in the RSNA and BP in NT rats.
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