The role of glutamate in opiate descending inhibition of nociceptive spinal reflexes
References (27)
- et al.
Dissociation of supraspinal and spinal actions of morphine: a quantitative evaluation
Brain Research
(1980) - et al.
Reversal of morphine and stimulation produced analgesia by subtotal spinal cord lesions
Pain
(1977) - et al.
Evidence that an excitatory connection between the periaqueductal gray and nucleus raphe magnus mediates stimulation-produced analgesia
Brain Research
(1979) - et al.
Evidence for opioid and non-opioid forms of stimulation-produced analgesia in the rat
Brain Research
(1982) - et al.
Acidic amino acid binding sites in mammalian neuronal membranes: their characteristics and relationships to synaptic receptors
Brain Res. Rev.
(1984) Opiate and opioid effects on brainstem neurons: relevance to nociception and antinociceptive mechanisms
Pain
(1982)The NMDA receptor: central role in pain inhibition in rat periaqueductal gray
Eur. J. Pharmacol.
(1988)- et al.
Spinal monoamine and opiate systems partly mediate the antinociceptive effects produced by glutamate at brainstem sites
Brain Research
(1984) - et al.
The antinociceptive role of a bulbospinal serotonergic pathway in the rat brain
Pain
(1988) - et al.
Site specificity in the development of tolerance to stimulation produced analgesia from the periaqueductal gray matter of the rat
Brain Research
(1987)
The effects of lesions of medullary midline and lateral reticular areas on inhibition in the dorsal horn produced by periaqueductal grey stimulation in the cat
Brain Research
(1984)
Nucleus raphe magnus lesions disrupt stimulation produced analgesia from ventral but not dorsal midbrain areas in the rat
Brain Research
(1983)
Characterization of inhibition of a spinal nociceptive reflex by stimulation medially and laterally in the midbrain and medulla in the pentobarbital-anaesthetized rat
Brain Research
(1984)
Cited by (0)
Copyright © 1990 Published by Elsevier B.V.