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Cited by (31)
Angiotensin converting enzyme and neprilysin inhibition alter pain response in dexhamethasone-induced hypertensive rats
2019, Pharmacological ReportsCitation Excerpt :It is uncertain whether decreased perception of pain is due to high blood pressure or both had a common mechanistic link. The hypoalgesia observed in chronic hypertension is associated with the changes in cholinergic, opioidergic, and noradrenergic functions [5]. Alterations of supraspinal and/or spinopetal pain modulatorys systems through sino-aortic afferents secondary to increased systemic arterial blood pressure may be a one the possible explanation of hypoalgesia observed in hypertensives [6].
Role of μ-opioid receptors in insulin release in the presence of inhibitory and excitatory secretagogues
2002, European Journal of PharmacologyBrainstem noradrenergic control of nociception is abnormal in the spontaneously hypertensive rat
2000, Neuroscience LettersThe role of dopamine in the nucleus accumbens in analgesia
1999, Life SciencesContribution of peripheral α<inf>1a</inf>-adrenoceptors to pain induced by formalin or by α-methyl-5-hydroxytryptamine plus noradrenaline
1996, European Journal of Pharmacology
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