Elsevier

Biochemical Pharmacology

Volume 41, Issue 10, 15 May 1991, Pages 1536-1537
Biochemical Pharmacology

Short communication
Protection by boswellic acids against galactosamine/endotoxin-induced hepatitis in mice

https://doi.org/10.1016/0006-2952(91)90575-PGet rights and content

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    They inhibit growth and affect apoptosis in brain tumors, colon cancer cells, breast and prostate cancers cells, malignant glioma cells, and leukemia cells [4–7]. Moreover, their biological activities against ulcerative colitis, asthma, chronic colitis, hepatitis, inflammation, and arthritis are well documented [8–13]. They also act as chronic inflammatory agents, mainly by blocking the activities of 5-lipoxygenase and different cytokines (interleukins and TNF-α) [14–17].

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    3-O-Acetyl-11-keto-β-boswellic acid (AK) is a pentacyclic triterpene obtained from gum resin of Boswellia serrata plant. Boswellia has a long history for being used in traditional Ayurvedic medicines for the treatment of inflammatory diseases (Safayhi et al., 1992; Singh et al., 1986), crohn disease, ulcerative colitis and arthritis (Gerhardt et al., 2001; Gupta et al., 2003; Kiela et al., 2005; Safayhi et al., 1991). Moreover, studies have also advocated the efficacy of AK in a variety of tumor cell lines (Liu et al., 2002; Park et al., 2002; Syrovets et al., 2005; Winking et al., 2000; Zhao et al., 2003).

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