Abstract
Presynaptic gamma-hydroxybutyric acid (GHB) and gamma-aminobutyric acidB (GABAB) receptor-mediated release of GABA and glutamate (GLU) in rat thalamic ventrobasal nucleus (VB): a possible mechanism for the generation of absence-like seizures induced by GHB.
P K Banerjee and O C Snead 3rd
Journal of Pharmacology and Experimental Therapeutics June 1995, 273 (3) 1534-1543;